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Dual specificity phosphatase 1 regulates human inducible nitric oxide synthase expression by p38 MAP kinase. Impaired inflammatory and pain responses in mice lacking an inducible prostaglandin E synthase. Anti-inflammatory effects and molecular mechanisms of loquat Eriobotrya japonica tea. Adiponectin stimulates prostaglandin E 2 production in rheumatoid synovial fibroblasts. SP, an anthrapyrazolone inhibitor of Jun N-terminal kinase.

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Microsomal Prostaglandin E Synthase-1 in Rheumatic Diseases

However, as also demonstrated inhibitors with potencies to both rodent and human species exist Pawelzik et al. In addition, the suppression of mPGES-1 may require higher glucocorticoid concentration, as previously observed after intra-articular treatment with high doses of glucocorticoids Korotkova et al. Author information Article notes Copyright and License information Disclaimer. Superinduction of cyclooxygenase-2 activity in human osteoarthritis-affected cartilage. Membrane-bound prostaglandin E synthasemediated prostaglandin E2 production by osteoblast plays a critical role in lipopolysaccharide-induced bone loss associated with inflammation.

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Shown is a representative gel of nine with similar results. The expression of cPGES was also observed in synovial lining cells, sublining mononuclear and fibroblast-like cells, mononuclear infiltrates, and some blood vessels Westman et al. Membrane-associated prostaglandin E synthase-1 is upregulated by proinflammatory cytokines in chondrocytes from patients with osteoarthritis. Effects of immunosuppressive treatment on microsomal prostaglandin E synthase 1 and cyclooxygenases expression in muscle tissue of patients with polymyositis or dermatomyositis. Thus, overexpression of mPGES-1 in OA synovial tissue and cartilage might contribute to chronic inflammation, pain, and catabolic processes related to OA.

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